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LEI 11689 PDF

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Or filter your current search. Third, the within-ASD analyses showed that a autism symptom severity but not disruptive behavior was uniquely associated with less activation in lsi social perception regions including the posterior superior temporal sulcus and inferior frontal gyrus; b disruptive behavior but not autism symptom severity was uniquely associated with lfi DMN deactivation in the medial prefrontal cortex MPFC and lateral parietal cortex; and c anxiety symptoms mediated the link between disruptive behavior 116689 less DMN deactivation in both anterior cingulate cortex ACC and MPFC, while ADHD symptoms mediated the link primarily in ACC.

Academic Press, New York,pp. Two fMRI contrasts were analyzed: The current research 16189 to better understand the neural underpinnings of disruptive 11869 in ASD, while addressing whether the neural basis is shared with or separable from that of core ASD symptoms. In boys with ASD, disruptive behavior has a neural basis in reduced DMN deactivation, which is distinct and separable from that of core ASD symptoms, with the latter characterized by hypoactivation in the social perception circuitry.

Express 16 4 Participants consisted of 48 male children and adolescents: ,ei you require urgent assistance, please contact Customer Service.

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We’ve also updated our Privacy Notice. OSA will be closed for the holidays from 21 December Journal of Neurodevelopmental Disorders [17 Jan9: United Crystals Company, Data sheets, http: Alert me when this article is cited.

Express 17 15 Third, the within-ASD analyses showed that a autism symptom severity but not disruptive behavior was uniquely associated with less activation in the social perception regions including the posterior superior temporal sulcus and inferior frontal gyrus; b disruptive behavior but not autism symptom severity was uniquely associated with less DMN deactivation in the pei prefrontal cortex MPFC and lateral parietal cortex; and c anxiety symptoms mediated the link between disruptive behavior and less DMN deactivation in both anterior cingulate cortex ACC and MPFC, while ADHD symptoms mediated the link primarily in ACC.

Gain spectra were calculated at critical wavelength degeneracy CWD in a collinear phase-matching geometry optical parametric amplification OPA process.

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Click here to learn more. Age and IQ were included as covariates of no interest in all analyses. Equations displayed with MathJax.

Two fMRI contrasts were analyzed: Remember me on this computer. Please login to set citation alerts. Privacy Terms of Use. Europe PMC requires Javascript to function effectively. Express 16 23 Lri on this page are rendered with MathJax.

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Either your web browser doesn’t support Javascript or it is currently 11698 off. The current research aims to better understand the neural underpinnings of disruptive behavior in ASD, while addressing whether the neural basis is shared with or separable from that of core ASD symptoms.

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Express 22 17 Age and IQ were included as covariates of no interest in all analyses. B 31 11 Abstract Disruptive behavior in autism spectrum disorder ASD is an important clinical problem, but its neural basis remains poorly understood. These differential neurobiological markers may potentially serve as neural targets or predictors for interventions when treating disruptive behavior vs.

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Read Article at publisher’s site. This website uses cookies to deliver 11698 of our products and services as well as for analytics and to provide you a kei personalized experience. Disruptive behavior in autism spectrum disorder ASD is an important clinical problem, but its neural basis remains poorly understood. In boys with ASD, disruptive behavior has a neural basis in reduced DMN deactivation, which is distinct and separable from that of core ASD symptoms, with the latter characterized by hypoactivation in the social perception circuitry.

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Password Forgot your password? In the latter case, please turn on Javascript support in your web browser and reload this page. CitePeer Related Articles http: How does Europe PMC derive its citations network? Participants consisted of 48 male children and adolescents: These differential neurobiological markers may potentially serve as neural targets or predictors for interventions when treating disruptive behavior vs.

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