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Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might eventually be mediated by modulation of MAPK signaling cascades. Here, we uncovered the therapeutic effect of morin against osteoporosis and demonstrated its suppressive action on the MAPK pathway in this disease.
Gene Ontology GO Terms.
In the current study, we tested the hypothesis that Ginkgo biloba extract GBE prevented hyperinsulinism-induced glucose intolerance in hepatocytes.
Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription. Our lej suggest that GBE has the potential to prevent insulin resistance and is a promising anti-diabetic drug.
How does Europe PMC derive its citations network? Find all citations in this journal default. We investigated the effects of GBE on glucose consumption, glucokinase activity, and mRNA levels of key genes in glucose metabolism and the insulin signaling pathway. How does Europe PMC derive its citations network?
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Journal of Natural Medicines [03 Sep65 1: Abstract Our aim is to investigate the potential therapeutic value of morin against osteoporosis and elucidate the mechanism of action. It is said that GBE treatment could avoid drug-induced obesity.
No matching affiliation detected. Body mineral density BMD was determined at proximal femurs using dual energy X-ray absorptiometry.
Morin treatment also increased the number of trabecular bones in DEX-induced rats.
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CitePeer Related Articles ldi The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively. Or filter your current search.